Neuro Px Extra Strength 60 vegi caps 1192-U
Formulation Features
A unique blend of specific compounds focused on neurological health supporting cell preservation, cellular energy production, bile acid-mediated signaling, general metabolic health and helping to reduce neurological oxidative damage and cellular stress.
Because Tauroursodeoxycholic acid (TUDCA) is able to cross the blood brain barrier reaching neuronal tissues, acting as a chemical chaperone to support healthy endoplasmic reticulum function we included it in this formula.
Nicotinamide riboside, a precursor to NAD+(nicotinamide adenine dinucleotide) supports a natural pathway to NAD+. NAD+ is a coenzyme involved in many biological functions and is essential to the production of energy in the body.
Pterostilbene is a phenolic compound found in small berries and nuts including blueberries and grapes. Pterostilbene and resveratrol structurally resemble each other; however, pterostilbene has better bioavailability and is included in the blend to help reduce cellular stress and oxidative stress.
In addition, Ashwagandha is an adaptogen and supports mental well-being. Studies show it helps to support cognitive function and concentration. Preliminary research suggests that ashwagandha may be neuroprotective through mitigating the effects of stress and protecting the nervous system.
| Ingredients | Amount Per Serving | % Daily Value |
|---|---|---|
|
500 mg | †|
|
350 mg | †|
|
250 mg | †|
|
100 mg | †|
| †Daily Value (DV) not established | ||
Other Ingredients:Â Vegetable Cellulose (capsules)
Ingredient Science:Â Ashwagandha Monograph
Suggested Use:
Take two capsules daily. Take with meals to help with absorption and decrease the chance of stomach irritation.
Warning:Â Consult with your physician before use. Do not use products if you are pregnant, nursing or breastfeeding. Discontinue use and contact your doctor if any adverse reactions occur. Do not exceed dosage of four capsules a day.
This product helps with supporting healthy endoplasmic reticulum stress inhibition and neurological autophagy processes.





